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ATX-mediated relief of lidocaine inhibition is largely occluded by the β4 peptide. A, B, Sample traces from a CA3 neuron with the β4 peptide, in control (A) and in ATX (B), in 50 mm extracellular Na, with conditioning: thin black line, −30 mV; gray line, 0 mV; thick black line, +60 mV. C, Percentage lidocaine inhibition recorded without (up triangles, n = 5) and with (down triangles, n = 6) ATX versus delay of lidocaine onset relative to the conditioning step.
