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. Author manuscript; available in PMC: 2013 Jun 1.
Published in final edited form as: Hear Res. 2012 Feb 8;288(0):47–57. doi: 10.1016/j.heares.2012.01.011

Fig. 2.

Fig. 2

Projections to the DNLL and their influences. A: A Monaural tone at the contralateral ear evokes a sustained discharge that lasts for the duration of the tone burst. The circuit activated is shown at top left. B: Background discharges evoked by the iontophoretic application of a glutamate/aspartate cocktail. C: A monaural tone at the ipsilateral ear evokes an inhibition that outlasts the duration of the tone that evoked it by 60 ms. The inhibition is expressed by the gap in the background activity. D: Iontophoretic application of the glycine receptor antagonist, strychnine, blocks the inhibition for the duration of the tone burst but does not block the persistent inhibition. E: Iontophoretic application of the GABA receptor antagonist, bicuculline, together with strychnine, eliminates all of the sound evoked inhibition.