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. 2013 May;81(5):1471–1478. doi: 10.1128/IAI.00048-13

Fig 2.

Fig 2

Replication of wild-type B. mallei and B. mallei AHL synthesis mutants in RAW 264.7 macrophage-like cells. Monolayers were infected with the wild-type (WT) GB8, the bmaI1 mutant (CM8), the bmaI3 mutant (CM9), or the double bmaI1 bmaI3 mutant (CM38) at an MOI of 0.1. The AHL synthesis mutations did not influence initial attachment (data not shown). The amount of intracellular bacteria harvested from RAW 264.7 cells after 20 h is shown as total CFU count.