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. Author manuscript; available in PMC: 2014 May 1.
Published in final edited form as: J Cardiovasc Pharmacol. 2013 May;61(5):401–407. doi: 10.1097/FJC.0b013e31828748ca

Figure 1.

Figure 1

SIN-1 prolongs the atrial and ventricular action potential. (A) The peroxynitrite scavenger uric acid was used to evaluate specificity of SIN-1. Atrial action potentials were recorded at baseline, with uric acid and with uric acid + SIN-1. No statistical change in atrial APD90 was observed after co-superfusion. (B) SIN-1 prolongs the atrial action potential and (C) SIN-1 prolongs the action potential and decreases the phase 1 notch in ventricular myocytes.