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. 2013 Jan 7;54(3):453–466. doi: 10.1093/jrr/rrs128

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© The Author 2013. Published by Oxford University Press on behalf of The Japan Radiation Research Society and Japanese Society for Therapeutic Radiology and Oncology

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 2. — Delayed induction of γ-H2AX foci and chromosomal instability in post-irradiation thymocytes. Symbols are the same as those in Fig. 1 except that in (A), open squares and closed squares indicate individual irradiated mouse data and mean values ± standard deviations, respectively. Open circles and closed circles indicate individual control mouse data and mean values ± standard deviations, respectively. TL, thymic lymphoma. (A) Delayed induction of γ-H2AX foci after irradiation. (B) Delayed appearance of aneuploidy after irradiation. (C) Induction of aneuploidy by H₂O₂ in thymocytes. (D) Trisomy 15 in a thymocyte 8 weeks after irradiation. Bar, 10 μm. (E) Chromatid break (arrow) in a thymocyte 8 weeks after irradiation. Bar, 10 μm. (F) Delayed induction of chromosomal aberrations in post-irradiation thymocytes cultured with proliferative stimuli for 48 h. (G) Induction of chromosomal aberrations in vivo in thymocytes 8 weeks after irradiation.