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. Author manuscript; available in PMC: 2013 May 13.
Published in final edited form as: Eur J Neurosci. 2011 May 25;33(12):2228–2237. doi: 10.1111/j.1460-9568.2011.07706.x

Fig. 5.

Fig. 5

Activation of 5-HT2A receptors prevented the abolition of gasping in vitro. (A) Gasping was evoked after application of hypoxic aCSF. (B) Application of DOI (20 μM) prevented the effect of blocking α2-NRs in the transverse slice preparation. (C) Histogram showing fictive inspiratory activity during application of hypoxic aCSF (control, open bins). After application of DOI and yohimbine the VRG burst frequency was maintained during the last minutes of application of hypoxic aCSF (filled bins, n = 5). The onset of the 10-min application of hypoxic aCSF was at the origin.