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. 2012 Dec 6;15(5):742–758. doi: 10.1111/cmi.12069

Table 2.

Mean values and statistical significance of recordings with blunted voltage-operated Ca2+ channels (VOCC) and voltage-gated Na+ channels (VGNC)

8 nM HlgC/HlgB Mean [Ca2+] peak ± SEM Latency s ± SEM Pairwise comparison of mean latencies (Holm-Sidak method)
a: control (65 cells) 270 ± 15 nM 210 ± 20 s a versus b: P < 0.001; a versus c: P < 0.001; a versus d: P < 0.001; a versus e: P < 0.001
b: blockade of L-, N- and P/Q-Type VOCC by 5 μM nifedipine + 0.1 μM ω-GVI-A + 0.1 μM ω-Aga-TK (88 cells) 265 ± 12 nM 116 ± 4 s b versus c: P < 0.001; b versus d: P < 0.001; b versus e: P = 0.839
c: blockade of VGNC by 0.3 μM TTX (24 cells) 263 ± 22 nM 436 ± 8 s c versus d: P < 0.001; c versus e: P < 0.001
d: blockade of L- and N-Type VOCC, VGNC and Glu-R by 5 μM nifedipine + 0.1 μM ω-GVI-A + 0.3 μM TTX + 10 μM CNQX + 10 μM D-AP5 (26 cells) 196 ± 10 nM 853 ± 14 s d versus e: P < 0.001
e: blockade of L-, N- and P/Q-Type, VGNC and Glu-R: 5 μM nifedipine + 0.1 μM ω-GVI-A + 0.1 μM ω-Aga-TK + 0.3 μM TTX + 10 μM CNQX + 10 μM D-AP5; 21 cells 232 ± 14 nM 111 ± 6 s

Mean values and statistical significance of peak amplitudes and latencies of the experiment shown in Fig. 4. The pairwise comparison between the means of the peak amplitude did not reveal significant statistical differences, whereas mean latencies were significantly different, but for experiments b and e only.