Table 4. Additional genetic interactions of dom.
| Genotype | C96-domR (% of Nicking) | N Wings Scored |
|---|---|---|
| UAS- atg1 (Unc51) | E (86%) | 322 |
| atg6, TRiP | E (82%) | 2074 |
| atg7, TRiP | E (75%) | 1724 |
| atg8A, TRiP | E (73%) | 1645 |
| atg8B, TRiP | E (78%) | 2227 |
| atg12, TRiP | E (73%) | 2147 |
| atg9, TRiP | E (72%) | 1520 |
| atg5, TRiP | E (63%) | 1773 |
| atg4, TRiP | ne (58%) | 2049 |
| atg2, TRiP | ne (58%) | 1736 |
| atg18, TRiP | ne (55%) | 1845 |
| PP2A wdb7 Regulatory | E (70%) | 638 |
| PP2A wdb, TRiP Regulatory | E (91%) | 236 |
| PP2A tws, TRiP Regulatory | E (83%) | 1021 |
| PP2A wrd, TRiP Regulatory | E (68%) | 1748 |
| PP2A mts, TRiP Catalytica | E (100%) | 478 |
| PP2A 29B, TRiP Scaffolda | E (100%) | 350 |
| CK1, UAS-dcoK4 | E (90%) | 1028 |
| CK1, dco TRiP | S (40%) | 1390 |
Column labeled C96-domR shows the percentage of wing nicking from crosses of C96-domR to listed genotypes. All genotypes are UAS-regulated except for wdb7. The interaction between these genotypes and C96-domR is as described for Table 2. All wing nicking differences were highly significant (P < 0.001, chi-square test), except for those labeled ne (no effect). Regulatory, catalytic and scaffold indicate the encoded function within the PP2A phosphatase complex. E, enhancer; S, suppressor.
a
These strains exhibited wing nicking at high penetrance with control crosses to C96-GAL4; all other UAS strains showed normal wings in the control cross.