Skip to main content
. Author manuscript; available in PMC: 2014 Jun 1.
Published in final edited form as: Menopause. 2013 Jun;20(6):631–639. doi: 10.1097/GME.0b013e31827c5c45

Table 4.

Results reported from the final models of separate regression equations examining paths in the proposed meditational model to determine whether ovarian function indexed by change in menstrual cycle length mediates the relation between depression history and cardio-metabolic risk.a

Path A Path B Path C Path C′
Depressionb → Ovarian Fxc Ovarian Fxc → Cardio-metabolic Riskd Depressionb → Cardio-metabolic Riskd (unadjusted for ovarian function) Depressionb → Cardio-metabolic Riskd (adjusted for ovarian function)
b p b p b p b p
.990 .001 0.125 .040 0.152 .040 0.129 .083
a

Analyses included covariate-adjustment for age, race/ethnicity, SES, smoking, physical activity level, parity, past use of hormone-containing medication for birth control, menstrual cycle length, and AMH.

b

Depression was represented by the lifetime history of depression composite score coded having received a depression diagnosis or having used anti-depressant medications (yes/no).

c

Ovarian function was indexed by change in menstrual cycle length coded any change vs. no change.

d

Cardio-metabolic risk was represented by the cardio-metabolic risk composite score reflecting the mean of standardized values of 5 individual risk factors (HDL, triglycerides, waist circumference, glucose, hypertensive status) with HDL reversed.