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. 2013 Jun 1;24(11):1676–1687. doi: 10.1091/mbc.E12-10-0772

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© 2013 Dui et al. This article is distributed by The American Society for Cell Biology under license from the author(s). Two months after publication it is available to the public under an Attribution–Noncommercial–Share Alike 3.0 Unported Creative Commons License (http://creativecommons.org/licenses/by-nc-sa/3.0).

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FIGURE 6: — dSkp2 genetically interacts with dap and CycE in the Drosophila wing. (A) A control wing of en-Gal4. (B) dSkp2 knockdown in the posterior region of the wing under the control of en-Gal4 leads to the typical WHS phenotype. Introducing a mutant copy of either dap⁰⁴⁴⁵⁴ (C) or dap⁴ (C′) into the en>dSkp2^KK108837 flies partially suppresses the WHS phenotype. Overexpression of CycE from UAS-CycE (D, D′, two independent transgenes) almost completely rescues the WHS phenotype caused by en>dSkp2^KK108837.