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. 2013 Apr 9;108(10):2178–2185. doi: 10.1038/bjc.2013.155

Table 2. Glioma risk and rs78378222 stratified by morphology type: IDH1 mutation, EGFR amplification, CDKN2A-p16-INKa homozygous deletion, 9p and 10q loss, 1p-19q co-deletion status in the French case–control series.

  Non-GBM GBM
Tumour alteration
N
OR
95% CI
P-value
N
OR
95% CI
P-value
IDH1+
272
3.0
1.6–5.6
5.0 × 10−4
24
7.8
2.2–27.3
2.0 × 10−4
IDH1
204
1.4
0.5–3.5
0.52
238
2.6
1.3–5.3
4.7 × 10−3
EGFR amplified
35
1.6
0.2–12.0
0.65
81
1.4
0.3–5.8
0.66
EGFR nonamplified
438
2.5
1.4–4.4
1.5 × 10−3
185
3.8
1.9–7.4
4.1 × 10−5
p16 Deleted
73
3.2
1.1–9.1
0.03
97
3.6
1.5–8.8
2.9 × 10−3
p16 Nondeleted
393
2.2
1.2–4.0
0.01
164
2.8
1.3–6.1
8.0 × 10−3
9p Deleted
140
2.0
0.8–5.2
0.14
128
3.2
1.4–7.3
4.7 × 10−3
9p Nondeleted
320
2.5
1.3–4.7
3.8 × 10−3
135
3.0
1.3–6.9
7.3 × 10−3
10q Deleted
140
2.4
1.0–5.9
0.04
203
2.0
0.9–4.5
0.1
10q Nondeleted
316
2.3
1.2–4.5
9.0 × 10−3
62
6.9
2.9–16.3
3.4 × 10−7
1p-19q Codeleted
146
1.9
0.7–5.0
0.17
28
NA
NA
0.47
1p-19q Noncodeleted 316 2.7 1.5–5.0 1.1 × 10−3 235 3.4 1.8–6.5 1.0 × 10−4

Abbreviations: CI=confidence interval; GBM=glioblastoma multiforme; OR=odds ratio.The analysis is based on directly genotyped data for 739 cases and 1,827 controls. Per allele odds ratios are shown with the risk allele for all tests being the C-allele with the reference being AA homozygotes.