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. Author manuscript; available in PMC: 2013 Jun 3.
Published in final edited form as: Sci Signal. 2012 Jun 12;5(228):ra43. doi: 10.1126/scisignal.2002437

Fig. 1. Elimination of alg-1 enhances netrin-dependent axon attraction, suppressing AVM axon ventral guidance defects in slt-1 mutants.

Fig. 1

(A) Schematic drawing highlights signaling pathways that influence AVM axon ventral guidance in development. (B) Schematic diagram of wild-type and mutant AVM axons. (C) alg-1 mutation specifically suppressed AVM axon ventral guidance defects in slt-1 (slit) but not unc-6 (netrin) mutants. alg-1(gk214) and alg-2(ok304) used in this study are null alleles. dcr-1(mg375) is a reduction-of-function allele. Error bars represent standard error of the proportion. Asterisks and brackets represent P < 0.001 by Z-test for two proportions.