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. 2013 Jun 1;30(11):981–997. doi: 10.1089/neu.2012.2699

FIG. 3.

FIG. 3.

Enzyme-linked immunosorbent assay (ELISA) scatter plots of Aβ40 and Aβ42 levels detected in gray matter (GM) and white matter (WM) of the frontal lobe. (A) Flow chart summarizes GM and WM preparation for ELISA quantification. (B) GM Tris-soluble Aβ40. (C) GM Tris-soluble Aβ42. (D) GM GDFA/GHCl-soluble Aβ40. (E) GM GDFA/GHCl-soluble Aβ42. (F) WM GDFA/GHCl-soluble Aβ42. The WM of all dementia pugilistica and non-demented control (NDC) cases had no detectable levels of Aβ40 (data not shown). Notice that case #2, the only ApoE ɛ4 carrier, had the largest amount of Aβ40 and Aβ42 in the GM and WM and the concurrent diagnosis of Alzheimer disease. See Figure 2 for shape codes. GDFA/GHCl, glass-distilled formic acid/5 M guanidine hydrochloride.