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The hypothetic mechanisms by which TBHQ reduces brain Aβ load in AD. TBHQ increases antioxidant capacity and reduces oxidative stress levels in the brain, which leads to suppression of the expression of PAI-1, a redox regulated protein, and induction of LRP-1 and thereby increased Aβ degradation/efflux from the brain. Solid lines represent evidence demonstrated directly from the present study.
