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. Author manuscript; available in PMC: 2014 Jun 1.
Published in final edited form as: Biochim Biophys Acta. 2013 Mar 13;1833(6):1553–1561. doi: 10.1016/j.bbamcr.2013.03.001

Fig. 5.

Fig. 5

Full-length A1 and A1ΔC mutant protect HUVEC and HDMEC from cell death induced by staurosporine (STS). Non-transduced (NT), rAd.A1, rAd.A1ΔC, and rAd.βgaltransduced (A) HUVEC and HDMEC were treated with STS (100 nM) for 6 h, then labeled with propidium iodide. Apoptosis was evaluated by FACS analysis of DNA content. Data shown are representative of 3 independent experiments. The percentage of cells undergoing cell death, as evidenced by DNA<2N (subG1 phase) is reported. (B) Bar chart representation of mean percentage of cell death ± SEM of the 3 experiments performed. *p<0.05, **p<0.01, ***p<0.001 indicate significance among different treatment groups, i.e. non tranduced (NT) EC and EC transduced with different rAd. #p<0.05, ##p<0.01, ###p<0.001 indicate significance between non stimulated (NS) and STS-treated HUVEC and HDMEC within the same experimental group.