Table 11.

Antiatherosclerosis of salvianolic acid B in vitro.

Cells/tissues	Effects	Reference
VSMCs	Nrf2-ARE signaling pathway, activation of NQO1.	Hur et al., 2010 [71]
H-monDC	Supress activation of PPARγ nuclear translocation, expression of CD40, CD86, CD1a, and HLA-DR, expression of IL-12 and IL-10, production of TNF-α, regulation of TLR4, myeloid differentiation of primary reactive protein 88, p38-MAPK signaling pathway.	Sun et al., 2011 [72]
Macrophage	Reduce mLDL uptake, antagonize CD36.	Bao et al., 2012 [73]
HASMCs	MMPs protein synthesis, downregulate ERK1/2, downregulate c-JNK.	Lin et al., 2007 [74]

Note: H-monDC: human monocyte-derived dendritic cells; Nrf2: nuclear factor 2-related factor 2; NQO1: NAD(P)H quinine oxidoreductase-1; PARP: poly ADP-ribose polymerase; ARE: antioxidant responsive element; VSMC: vascular smooth muscle cells; IL: interleukin; TLR: toll like receptor; TNF: tumor necrosis factor.