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. Author manuscript; available in PMC: 2013 Jul 9.
Published in final edited form as: Am J Hypertens. 2013 Jan;26(1):95–103. doi: 10.1093/ajh/hps023

Table 4.

Multivariable-adjusted Correlations of the Carotid and Aortic AIs with Urinary Measurements

Carotid AI
Aortic AI
Correlates β ± SE P r2 β ± SE P r2
Longitudinal analysis
 Sodium concentration (+40 mmol/L) −1.38 ± 0.66 0.04 0.39 −1.54 ± 0.72 0.02 0.50
 Sodium excretion (+60 mmol) 0.10 ± 0.75 0.90 0.01 0.03 ± 0.81 0.96 0.03
 Creatinine concentration (+4 mmol/L) −0.30 ± 0.90 0.74 0.02 −1.22 ± 0.96 0.21 0.47
 Creatinine excretion (+4 mmol) 1.55 ± 1.14 0.17 0.28 0.92 ± 1.23 0.46 0.37
Cross-sectional analysis
 Sodium concentration (+40 mmol/L) −1.26 ± 0.70 0.07 0.26 −1.52 ± 0.76 0.04 0.32
 Sodium excretion (+60 mmol) 0.33 ± 0.67 0.62 0.25 0.57 ± 0.73 0.43 0.04
 Creatinine concentration (+4 mmol/L) −1.79 ± 0.82 0.02 0.41 −2.78 ± 0.89 0.001 0.96
 Creatinine excretion (+4 mmol) −0.77 ± 0.89 0.38 0.30 −0.95 ± 0.97 0.33 0.06

The AIs were standardized to a heart rate of 75 beats/min. All estimates were adjusted to the covariables are listed in Table 3. β ± SE, P, and r2 indicate the effect size ±SE, statistical significance, and the percentage variance of the dependent variable explained by a single covariable, respectively. Effect sizes are expressed for a ~1 SD higher value of the explanatory variables. Abbreviation: AI, augmentation index.