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. 2013 May 2;134(2):291–303. doi: 10.1093/toxsci/kft104

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Published by Oxford University Press on behalf of the Society of Toxicology 2013. This work is written by (a) US Government employee(s) and is in the public domain in the US.

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Fig. 4. — BDCM exposure and CYP2E1-dependent modulation of hepatic glucose and fat metabolism. (A) Liver mRNA expression of glucose transporter (Glut-1, Glut-4), glycolytic enzyme (PFK), and gluconeogenic enzyme (PCK-1) in DIO, DIO + BDCM, and CYP2E1 KO + BDCM mice (n = 3). (B) Liver mRNA expression of lipid metabolism master regulator, PGC-1α, lipogenic gene SREBP-1c, and regulators of lipid metabolism, PPAR-α and PPAR-γ in DIO, DIO + BDCM, and CYP2E1 KO + BDCM mice (n = 3). *p < 0.05 is considered statistically significant.