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Genetic interactions between TORC2 and the Swi1-Swi3 fork replication protection complex. A, mutations in tor1+ and swi1+ show epistasis with respect to spontaneous Rad52-YFP foci level. Strains growing logarithmically in rich medium were stained with Hoechst and imaged live. The percentage of cells containing Rad52-YFP foci was calculated as described in the legend to Fig. 3. B, Tor1 and Swi1 show additive sensitivity to replication stress.
