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. 2013 Aug 15;443(1):99–105. doi: 10.1016/j.virol.2013.04.028

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© 2013 Elsevier Inc.

Open Access under CC BY-NC-ND 3.0 license

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Fig. 3 — Replication kinetics of OUR T88/3 and recombinant OUR T88/3ΔDP2 viruses. Pig bone marrow macrophages were infected at a high multiplicity of infection (m.o.i.) of 10 or low m.o.i. of 0.1 with parental OUR T88/3 strain or recombinant virus OUR T88/3ΔDP2. At various hours post-infection, as indicated on the x axis, total virus was harvested and infectious virus titrated on 96 well plates by analysis of infection on cultures of pig bone marrow macrophages. The virus titre (TCID₅₀/ml) is the mean of three individual observations. Titres obtained following infection with viruses OUR T88/3 high m.o.i. and low m.o.i. ; OUR T88/3ΔDP2 high m.o.i. and low m.o.i. are indicated.

Inline graphic — Replication kinetics of OUR T88/3 and recombinant OUR T88/3ΔDP2 viruses. Pig bone marrow macrophages were infected at a high multiplicity of infection (m.o.i.) of 10 or low m.o.i. of 0.1 with parental OUR T88/3 strain or recombinant virus OUR T88/3ΔDP2. At various hours post-infection, as indicated on the x axis, total virus was harvested and infectious virus titrated on 96 well plates by analysis of infection on cultures of pig bone marrow macrophages. The virus titre (TCID₅₀/ml) is the mean of three individual observations. Titres obtained following infection with viruses OUR T88/3 high m.o.i. and low m.o.i. ; OUR T88/3ΔDP2 high m.o.i. and low m.o.i. are indicated.