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. Author manuscript; available in PMC: 2013 Jul 15.
Published in final edited form as: JAMA. 2009 Sep 9;302(10):1076–1083. doi: 10.1001/jama.2009.1295

Table 4.

Replication study: Characteristics of cystic fibrosis patients with (CFLD) and without (CF no LD) severe liver disease

Variable Replication study
CFLD
n = 136
CF no LD
n = 1088
Age (yrs)a
  Mean (± SD) 18.2 (± 6.2) 27.2 (± 9.2)
  Median 16.6 25.0
Gender (male) 82 (60.3%) 566 (52.0%)
Caucasian 125 (91.9%) 1066 (98.0%)
Genotype b
  PI/PI c 116 (85.4%) 1017 (93.5%)
  PI/PS c 4 (2.9%) 13 (1.2%)
  PS/PS 0 (0.0%) 2 (0.2%)
  PI/unknown 14 (10.3%) 44 (4.0%)
  unknown/unknown 2 (1.4%) 12 (1.1%)
Meconium ileus d 31 (23.8%) 62 (18.3%)
Age of diagnosis of portal hypertension (yrs) e
  Mean (± SD) 11.0 (± 4.7) N/A
  Median 11 N/A
  Range 0.5 – 28 N/A
Portal hypertension documented by f 
  Splenomegaly 124 (91.1%) N/A
  Varices (esophageal, rectal) 101 (74.2%) N/A
  Hypersplenism g 52 (44.4%) N/A
a

Age at time of enrollment.

b

CFTR mutations for CFLD patients in replication study: DF508/DF508 45.6%; DF508/PI 32.4%; DF508/unknown 9.6%; PI/PI 7.4%; PI/unknown 0.7%; PI/PS 2.9%; unknown/unknown 1.4%. CFTR mutations for CF no LD patients in replication study: DF508/DF508 62.8%; DF508/PI 27.5%; DF508/PS 0.6%; DF508/unknown 3.7%; PI/PI 3.1%, PI/unknown 0.4%, PI/PS 0.6%; PS/PS 0.2%; unknown/unknown 1.1%. See eTable 1 for specific CFTR genotypes for CFLD patients.

c

PI = Pancreatic exocrine insufficient mutation, PS = Pancreatic exocrine sufficient mutation.

d

Data available from 130 CFLD patients (ages 0–28 years) and 339 CF no LD patients (ages 15–28 years).

e

Data available from 120 CFLD patients.

f

Documented using several different imaging techniques; some patients had portal hypertension confirmed by more than one method; all patients tested had findings compatible with multi-lobular cirrhosis.

g

As defined by platelet count < 100,000/ul; data available on 117 patients.