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. 2011 May 4;31(18):6858–6870. doi: 10.1523/JNEUROSCI.0710-11.2011

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Copyright © 2011 the authors 0270-6474/11/316858-13$15.00/0

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Figure 3. — MTM, SK, and SDK protect immature primary cortical neurons (E17) from oxidative stress in a DNA binding-dependent manner. A, MTM, SK, and SDK abrogate neuronal cell death induced by HCA (5 mm) in a dose-dependent manner. SDK was protective at lower doses compared with MTM and SK. In addition, SK and SDK treatment induced no significant toxicity compared with non-HCA control, whereas MTM treatment did. *, Significant death compared with non-HCA control; **, significant protection compared with HCA treatment alone. PreB did not protect primary neurons from HCA-induced cell death. B, Live/Dead staining of treated neuron cultures. Live cells are identified by green, whereas dead cells are identified by red fluorescence.