Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2013 May 1;38(9):1816–1824. doi: 10.1038/npp.2013.81

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

Copyright © 2013 American College of Neuropsychopharmacology

PMC Copyright notice

PKA phosphorylation is decreased in the medial prefrontal cortex (mPFC), but not in the amygdala (amy), of PCP-treated rats. Effects of PCP on PKA expression (normalized to β-actin; center panel) and phosphorylation (pPKA; normalized to PKA; right panel) in the (a) amygdala (F_1,10=11.01, p<0.01 and F_1,11=2.04, NS for PKA and pPKA, respectively) and (c) mPFC (F_1,11=4.19, p=0.065 and F_1,10=16.63, p<0.01 for PKA and pPKA, respectively). Left panels show representative western blot data. Values are expressed as percentage of control (mean±SEM; n=6–7 per group). *p<0.05 compared with saline-treated rats. (b) Correlation analysis of PKA and AEA levels in the amygdala. (d) Correlation analysis of pPKA and AEA levels in the mPFC.