Figure 6.

Possible heat-stress-tolerance system in S. splendens. Initial effects of heat stress are on the plasmalemma and cause electrolyte leakage, ROS production, and oxidative damage. Signaling caused by these effects at the genomic level can lead to increased production of antioxidants and osmolytes that correct for perturbations in the cell-water balance. Production of protective enzymes and antioxidants results in less oxidative damage. The majority of the identified proteins are involved in photosynthesis, metabolism, protein processing, and stress response, indicating that many processes work together to reestablish cellular homeostasis after heat stress.