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. Author manuscript; available in PMC: 2013 Jul 25.
Published in final edited form as: J Cell Biochem. 2010 Apr 1;109(5):1033–1044. doi: 10.1002/jcb.22486

Fig. 3.

Fig. 3

A: Western blot and densitometry analysis of HO-1, HO-2 and β-actin expression in myocardium of control and diabetic mice untreated or treated with CoPP. **P< 0.01 diabetes versus control, §§P<0.01 diabetes CoPP versus diabetes. Histograms are mean ±SE, n = 4 in each group. B: Myocardial O2− production in control, diabetic mice, and diabetic mice treated with CoPP. *P< 0.05 diabetes versus control; §P<0.05 diabetes CoPP versus diabetes. Results are mean ±SE; n =4 in each group. C: Serum adiponectin levels in control and diabetic mice, untreated and treated with CoPP. Serum samples were obtained immediately prior to sacrifice. **P<0.01 diabetes versus control; §§P< 0.01 diabetes CoPP versus diabetes. Results are mean ±SE, n = 4 in each group.