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. 2013 May 21;6:25. doi: 10.1186/1756-6606-6-25

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Copyright © 2013 Hiraoka et al.; licensee BioMed Central Ltd.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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The development of BG was normal in Dll1^(loxP/loxP)/Ptf1a-Cre mice. (A, B) Immunohistochemistry for BLBP (green) in Dll1^(loxP/+)/Ptf1a-Cre (A) and Dll1^(loxP/loxP)/Ptf1a-Cre (B) mice at P7. (C) Quantification of the number of BLBP-positive cell bodies. (D, E) BG labeled by anti-GFAP (green) antibody in Dll1^(loxP/+)/Ptf1a-Cre (D) and Dll1^(loxP/loxP)/Ptf1a-Cre (E) mice at P7. (F, G) In situ hybridization for GLAST in Dll1^(loxP/+)/Ptf1a-Cre (F) and Dll1^(loxP/loxP)/Ptf1a-Cre (G) mice at P7. (H) Quantification of the number of GLAST-positive cells in EGL at P7. n.s., no significant. Scale bars: 50 μm (A, B, D, E), 250 μm (F, G). EGL, external granular layer; PCL, Purkinje cell layer.