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Despite constant susceptibility of intrasplenic MDSCs to sunitinib at MTD 40 mg/kg (D), intratumoral MDSC resistance to sunitinib was evident to a varied but sometimes profound degree (E). For the 3 tumor models compared, the disparate susceptibility of tumors to 10 days of sunitinib treatment (regression, stabilization or progression, A–C) was predictive of intratumoral (D) but not intrasplenic MDSC susceptibility (E).
