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. 2013 Aug 6;7:395. doi: 10.3389/fnhum.2013.00395

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Copyright © 2013 Bigler.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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TOP: Fluid attenuated inversion recovery (FLAIR) sequence in three traumatic brain injury (TBI) cases depicting different levels of white matter burden. (Left) a child with mild TBI (mTBI) indicating a solitary, focal white matter hyperintensity (WMH). (Middle) a 62-year-old male with a severe TBI with no white matter abnormalities noted on admission CT. Patient had a GCS of 7 prior to intubation, meeting criteria for severe TBI (Right) 17-year-old injured 2 years prior with an admission GCS of 3. Note the prominent and extensive WMHs widely distributed. BOTTOM: The middle and right hand subjects are the same as above, but subject on the left side is a different child with a mild TBI, who did not have a WMH, but did show hemosiderin in the corpus callosum (arrow). Note that both patients with severe injury have some generalized atrophy and ventricular dilation as a reflection of generalized brain volume loss as a consequence of severe TBI along with multiple hemosiderin deposits.