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π cell fate defects following knockdowns of hda-1, nhr-67, and egl-43. π cells were counted in the early to mid-L4 stages in single and double RNAi-treated animals. The percentage of animals is plotted. nhr-67 and egl-43 suppress the extra π cell phenotype caused by the reduction of hda-1 function. The number of animals in each case (N) is shown.
