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. Author manuscript; available in PMC: 2014 Feb 1.
Published in final edited form as: Pediatr Res. 2013 Jun 3;74(2):127–132. doi: 10.1038/pr.2013.90

Figure 4.

Figure 4

The dependence of human milk fat digestion on the combined action of gastric lipase, CEL and PLRP2. In each case, 25 μg of each lipase and 5 μg of colipase were added to the incubation except that no colipase was added during the pre-incubation with gastric lipase. The assays were done on heat-treated human milk with a physiological mixture of bile salts (4 mM). The values are the mean ± SD (n = 3). (No gastric lipase) White bar, human milk; Gray bar, CEL; Coarse hatched bar, PLRP2; Fine hatched bar CEL & PLRP2. There is a statistically significant difference among the mean values of the different conditions as determined by one-way ANOVA (F(3,8)= 12.63, P = 0.002). *P < 0.05 PLRP2 (0.36 ± 0.2) CEL (0.39 ± 0.03) and CEL & PLRP2 (0.65 ± 0.1) compared to mother’s milk by the Holms-Sidak post-hoc pairwise multiple comparisons method. (Gastric lipase) White bar, human milk; Black bar, gastric lipase; Gray bar, CEL; Coarse hatched bar, PLRP2; Fine hatched bar, CEL & PLRP2. There is a statistically significant difference among the mean values of the different conditions as determined by one-way ANOVA (F(4,10)= 270.15, P = <0.001). *P < 0.05 CEL (0.7 ± 0.1) compared to mother’s milk (0.03 ± 0.03), **P < 0.05 PLRP2 (2.0 ± 0.2) compared with CEL, gastric lipase and mother’s milk, †P < 0.05 CEL & PLRP2 (4.5 ± 0.3) compared with PLRP2, CEL, gastric lipase and mother’s milk by the Holms-Sidak post-hoc pairwise multiple comparisons method.