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. 2010 Jun 21;12(5):690–696. doi: 10.1038/aja.2010.37

Table 3. Analysis of genotype frequencies of NOS2 gene polymorphisms, based on low and high PSA level, in subjects with BPH.

SNP (locus) Genotype PSA level (ng mL−1)
Model OR (95% CI) P-value
< 1.5
≥ 1.5
n = 84 (%) n = 144 (%)
rs2779248 T/T 70 (83.3) 111 (77.1) Codominant 1.26 (0.71–2.24) 0.430
  Promoter T/C 11 (13.1) 29 (20.1) Dominant 1.50 (0.74–3.01) 0.250
  −278 C/C 3 (3.6) 4 (2.8) Recessive 0.71 (0.15–3.33) 0.670
rs10459953 G/G 23 (27.4) 43 (29.9) Codominant 0.98 (0.68–1.44) 0.940
  5′UTR C/G 42 (50.0) 66 (45.8) Dominant 0.88 (0.48–1.62) 0.680
  C/C 19 (22.6) 35 (24.3) Recessive 1.10 (0.58–2.10) 0.760
rs2297518 G/G 63 (75.0) 104 (72.2) Codominant 1.19 (0.68–2.08) 0.540
  Missense A/G 20 (23.8) 36 (25.0) Dominant 1.16 (0.63–2.17) 0.630
  Ser608Leu A/A 1 (1.2) 4 (2.8) Recessive 2.05 (0.22–18.92) 0.500

Abbreviations: 5′UTR, 5′-untranslated region; BPH, benign prostatic hyperplasia; CI, confidence interval; n, number of subjects; NOS2, nitric oxide synthase 2; OR, odds ratio; PSA, prostate-specific antigen; SNP, single nucleotide polymorphism.

Note: P-values were obtained from logistic regression analyses with the codominant, dominant and recessive models without Bonferroni correction.