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. Author manuscript; available in PMC: 2013 Aug 10.
Published in final edited form as: Am Heart J. 2011 Apr 6;161(6):1038–1045. doi: 10.1016/j.ahj.2011.02.007

Figure 2.

Figure 2

Primary endpoint by etiology and SRS Kaplan-Meier curves for the primary endpoint stratified by HF etiology and SRS. Patients with a nonischemic etiology and a low SRS had the lowest event rate. Those with a high SRS, regardless of etiology, had similar event rates, with the subjects with nonischemic HF having slightly higher rates. Patients with an ischemic etiology and a low SRS had the highest event rate, likely from a high underlying ischemic burden.