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Trpm7 deletion in embryonic myocardium disrupts cardiac automaticity in vitro. (A) Ca2+ transients measured by high-speed line-scanning confocal microscopy in Trpm7fl/fl EVM 4–5 d after transduction with Ad-CMV-Lacz (Top), Ad-CMV-Cre (Middle), and Ad-TnT-Cre (Bottom). (B) Mean Ca2+ transient frequency, (C) cycle length, CL, (D) peak Ca2+ transients, F/Fo, and (E) Ca2+ transient length in Ad-CMV-Lacz, Ad-CMV-Cre, and Ad-TnT-Cre treated Trpm7fl/fl EVMs. *P < 0.05, **P < 0.01.
