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. 2001 Jul 17;98(15):8196–8202. doi: 10.1073/pnas.121008898

Figure 2.

Figure 2

The average number of UV-induced lesions that results in lethality is plotted for each E. coli strain. (A) Focusing on the question, Can recombination promote recovery following UV irradiation?, it has been suggested that because a uvrA mutant is more resistant to UV irradiation than a uvrArecA mutant, recA may promote survival in the uvrA mutant by recombining around DNA lesions. (B) However, focusing on the question, How do cells recover following UV irradiation?, one might conclude that the recombination does not significantly contribute to the recovery seen in wild-type cells (lesion numbers plotted: uvrA, 60; recA, 22; uvrArecA, 1.5; wild type, 3,600; as reported in ref. 28).