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Hypertension in response to placental ischemia
Hypertension in response to placental ischemia proceeds via immune activation, CD4+ T-cells mediating the release of angiotensin II type-1 receptor autoantibody (AT1-AA), and inflammatory cytokines that contribute to the increased vasoactive peptide ET-1 increased sensitivity to ANGII, oxidative stress, and sFlt-1, all known players in the pathophysiology of preeclampsia.
