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Proposed model of TGF-β inhibition of HPV16 URR activity through NFI-Ski interactions in TGF-β-sensitive HKc/HPV16. (A) In the absence of TGF-β NFI-Ski complexes bind to and activate the HPV16 URR promoter. (B) In the presence of TGF-β signaling, Ski is degraded and no longer available to complex with NFI to induce promoter activity of the HPV16 URR.
