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Inhibiting IL-15 signaling with TMβ1 delayed the diabetes development in NOD mice. (A) IL-15Rα expression on the purified islet cells from NOD mice. The histograms were gated on CD45− population. Gray, isotype control; solid, IL-15Rα. (B) The percentage of diabetic mice in NOD mice treated with PBS (n = 25) or monoclonal anti-CD122 antibody TMβ1 (n = 25) (P < 0.0001).
