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. Author manuscript; available in PMC: 2014 Sep 1.
Published in final edited form as: Lung Cancer. 2013 Jun 24;81(3):347–353. doi: 10.1016/j.lungcan.2013.05.011

Figure 2.

Figure 2

Association of Gender, Race and EGFR or KRAS positivity by Smoking Status. Females are represented by blue bars and males are represented by green bars. 2A. Caucasian female non-smokers had higher rate of EGFR positivity compared to African American female non-smokers, which did not meet statistical significance (44.4% (20/45) vs. 22.2% (2/9), p=0.28). Caucasian male non-smokers had a higher rate of EGFR positivity compared to African American male non-smokers; this difference did not meet statistical significance due to small number of African American never-smokers (39.1% (9/23) vs. 0% (0/1), p=1.00). 2B. Caucasian female smokers had a significantly higher rate of EGFR positivity than African American female smokers (11% (20/182) vs. 0% (0/31), p=0.05). Caucasian male smokers had a similar rate of EGFR positivity as African American male smokers (3% (4/135) vs. 4.5% (1/22), p=0.54). 2C. Caucasian female never-smokers had higher rate of KRAS mutant positivity compared to African American female never-smokers; this difference did not meet statistical significance (7.1% (2/28) vs. 0% (0/8), p=1.0). Caucasian male non-smokers had a higher rate of KRAS mutant positivity compared to African American male never-smokers, which did not meet statistical significance due to small number of African American never-smokers (18.8% (3/16) vs. 0% (0/1), p=1.00). 2D. Caucasian female smokers had a similar rate of KRAS mutant positivity as African American female smokers (42.5% (57/134) vs. 41.7% (10/24), p=1.0). Caucasian male smokers had a similar rate of KRAS mutant positivity as African American male smokers (22.8% (23/101) vs. 15.8% (3/19), p=0.76).