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. 2013 Jan 9;33(2):631–640. doi: 10.1523/JNEUROSCI.3936-12.2013

Figure 8.

Figure 8.

NMDAR–GABAAR crosstalk is blunted in rats with HF. A1, A2, Representative traces showing lack of IGABAA potentiation following activation of NMDARs either with NMDA (30 μm, A1), or by endogenous glutamate following GLT1 blockade with DHK (500 μm, A2). B, Summary data showing mean IGABAA charge transfer density before and after NMDAR activation in sham and HF rats (n = 18 and 13, respectively). C, Summary data showing lack of effects of intracellular dialysis with OKA, H-9, or ATPγS on the blunted NMDA-mediated IGABAA potentiation in MNCs from HF rats (n = 13, 20, 13, and 6, respectively). *p < 0.01 versus control.