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. 2013 May 24;1(1):292–296. doi: 10.1016/j.redox.2013.04.003

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Fig. 1 — Xanthine oxidase-derived O₂^•− impairs endothelial-dependent relaxation of rabbit aorta. (A) Dose-dependent relaxation to endothelium-dependent dilator acetylcholine (ACh) in the absence or presence of increasing concentrations of XO as indicated. (B) Dose-dependent relaxation to endothelium-independent dilator sodium nitroprusside (SNP) in the absence (black line) or presence (blue line) of 5 U/L XO. (n=8–12) ^⁎p<0.05 vs. control (no XO). (For interpretation of the references to color in this figure legend, the reader is referred to the web version of this article.)