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. 2013 May 29;6(5):1246–1259. doi: 10.1242/dmm.012005

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© 2013. Published by The Company of Biologists Ltd

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/3.0), which permits unrestricted use, distribution and reproduction in any medium provided that the original work is properly attributed.

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Fig. 4. — Vascular phenotype of notch3 mutants. (A–D) Lateral views of (A) notch3^zm/+ heterozygotes, (B) notch3^zm/zm mutants, (C) notch3^st51/+ heterozygotes and (D) notch3^st51/st51 mutants. (B) notch3^zm/zm mutant adults show craniofacial defects (black arrow) and accumulation of blood (light and dark blue arrows and arrowheads) in the head, pectoral anal and caudal fins. (E) Quantification of the bleeding phenotypes. Degree of shading indicates penetrance of phenotype: white cell in the table indicates less than 50% of animals showed the phenotype, light gray indicates that 50–75% of individuals were affected, and dark gray denotes greater than 75% of animals had the phenotype.