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. 2013 Sep 5;9(9):e1003736. doi: 10.1371/journal.pgen.1003736

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This is an open-access article distributed under the terms of the Creative Commons Public Domain declaration, which stipulates that, once placed in the public domain, this work may be freely reproduced, distributed, transmitted, modified, built upon, or otherwise used by anyone for any lawful purpose.

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A. Induction of G-C to A-T transitions by the mechanism of HAP misincorporation. B. Induction of A-T to G-C transition mutations by the mechanism of misincorporation opposite HAP. C. Induction of G-C to A-T mutations by cytosine deamination followed by incorporation of adenine opposite uracil. D. Numbers of SNVs induced by HAP in haploid (LAN201-1 - LAN201-4) and diploid (LAN211-1 – LAN211-10) strains. The proportions of substitution types are shown by color. E. Numbers of SNVs induced in haploid (LAN200-L1 - LAN200-L4) and diploid (LAN210-L1 – LAN210-L7), PmCDA1-induced mutants CAN^R mutants, and diploid PmCDA1-induced FOA^R mutants (LAN210-FOA-1 and LAN210-FOA-2). Proportions of substitution types are shown by color.