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. 2013 Jul 24;288(36):26112–26120. doi: 10.1074/jbc.M112.396648

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© 2013 by The American Society for Biochemistry and Molecular Biology, Inc.

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FIGURE 6. — The restoration of neuronal excitability by D₄ in stressed animals depends on the bi-directional regulation of CaMKII activity. A and C, representative spontaneous action potential recordings showing the effect of PD168077 (40 μm) in PFC neurons infused with active CaMKII versus GST from acutely stressed rats (A) or KN-93 versus KN-92 from repeatedly stressed rats (C). Scale bars, 20 mV, 2 s. B and D, scatter plots of firing rates before (con) and after PD168077 in the presence of various reagents affecting CaMKII activity in PFC pyramidal neurons from acutely stressed (B) or repeatedly stressed (D) rats. *, p < 0.01, t test.