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A hypothetical model for changes in mitochondrial angiotensin receptors with aging. Note that with aging the balance is tipped toward more expression of mtAT1Rs and less mtAT2Rs. The imbalance between mtAT1Rs and mtAT2Rs with aging may accelerate the development of age related oxidative stress, mtDNA mutations, apoptosis and senescence. ACEi, ACE inhibitor; ARBs, Ang II receptor blockers.
