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. 2013 May 27;2:746–758. doi: 10.1016/j.nicl.2013.05.008

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© 2013 The Authors

This is an open-access article distributed under the terms of the Creative Commons Attribution-NonCommercial-No Derivative Works License, which permits non-commercial use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig. 1 — A: Reductions in glucose utilization (red) and gray matter density (blue) in dementia syndromes in comparison with control subjects and each other, rendered onto MNI template. Overlap is in purple. Only clusters with p < 0.05 (corrected) are displayed. Color spectrum represents t values. Left is left. B: The bar chart illustrates the extension of hypometabolic and atrophic brain regions (sum of all clusters in voxels exceeding an uncorrected threshold of p < 0.001 on the voxel level, and p < 0.05 corrected on the cluster level). AD = Alzheimer's disease, C = control subjects, FTLD = frontotemporal lobar degeneration, MNI = Montreal Neurological Institute, n.s. = not significant.