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ELISA evaluations of TNFα and MCP-1 in PBMCs (A and B) or brain homogenates (C and D). 6-OHDA lesions increased TNFα in both PBMCs and brain homogenates (+p < 0.05 vs vehicle/sham) and treatment with AC-186 blocked this increase (*p < 0.05 vs vehicle/6-OHDA lesion). MCP-1 was increased by 6-OHDA in (B) PBMCs but not (D) brain (+p < 0.05 vs vehicle/sham), and the increase in PBMCs was not prevented by AC-186.
