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. 2013 Sep 24;7:161. doi: 10.3389/fncel.2013.00161

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Copyright © Persson, Lindberg and Kuhn.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Decreased phosphorylation of radixin in the RMS after DX52-1 treatment. (A) Merged image of radixin and phosphorylated rzrin/radixin/moesin (pERM) immunoreactivity in a coronal section of the control RMS. (B) pERM immunoreactivity in the control RMS. (C) Merged image of radixin and phosphorylated ezrin/radixin/moesin (pERM) immunoreactivity in a coronal section in the DX52-1 treated RMS. (D) pERMimmunoreactivity in the DX52-1 treated RMS. Radixin/pERM immunopositive circular structures in B and D are likely residual ependymal cells originating from the wall of the collapsed olfactory ventricle (Peretto et al., 1997). Ependymal cells are known to express high levels of ezrin which explains sustained pERM expression in this area in both control and DX52-1 treated RMS. Scale bar = 50 μm.