Skip to main content
. 2013 Sep 24;8(9):e75632. doi: 10.1371/journal.pone.0075632

Figure 4. dpn and E(spl) genes may act redundantly.

Figure 4

(A–C) The mild wing vein phenotype of E(spl)mγ-mβ (A) is dominantly modified by dpn[1] (B and C). (B) Arrows point to vein thickening at the tips of L4 and L5, observed in 74% of dpn[1]/+ E(spl)mγ-mβ wings (C) 26% of dpn[1]/+ E(spl)mγ-mβ wings also showed ectopic veins surrounding L5 (arrow). See Table S2. (D–E) E(spl)mβ (E-E′) or both dpn and E(spl)mβ (D-D′) levels were knocked down by RNAi at early third instar wing discs. Cut expression (green) was dramatically reduced in the double RNAi (D′) compared to E(spl)mβ-RNAi alone (E′). (F-F′) E(spl)-C loss of function MARCM clones produce reduction in Cut expression (F′) when combined with dpn RNAi.