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. Author manuscript; available in PMC: 2014 Nov 1.
Published in final edited form as: J Psychiatr Res. 2013 Aug 8;47(11):1630–1635. doi: 10.1016/j.jpsychires.2013.07.012

Figure 2.

Figure 2

Deficits in auditory information processing seen in the orbitofrontal cortex of MAM rats was not seen in the prelimbic and infralimbic cortices. However, the prelimbic cortex appeared to play a role in intensity processing at a greater latency than that seen in the orbitofrontal cortex. Both the prelimbic and infralimbic cortices display a prepulse intensity-dependent modulation of the response to the startle stimulus. (Top traces of each subfigure) Grand averaged AEPs for SAL and MAM treated animals respectively. (A,D) Earlier occuring maximum evoked potential amplitudes and (B,E) later occuring maximum evoked potential amplitudes for the prepulse intensities. (C,F) Evoked potential amplitudes of the P20 component of the potentials evoked to the startle-stimulus during both prepulse and startle alone trials (+0dB). * indicates a significant effect of treatment between SAL and MAM animals. # indicates a significant effect of prepulse intensity. Data are plotted as the mean ± SEM.