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. Author manuscript; available in PMC: 2013 Oct 7.
Published in final edited form as: Brain. 2008 Jul 16;131(Pt 8):2042–2060. doi: 10.1093/brain/awn145

Table 2.

Characteristics of patients: anatomical MRI features and EEG abnormalities

Pts Anatomical MRI Interictal events
Ictal events
Types Number Types Number Duration (mean; range)
1 R TP nodular heterotopia (trigone, occipital horn and subcortical region). Thick overlying cortex R T spike and waves 13 R FT sharp waves followed by polyspikes
Electrical events during sleep
24 3.5 s; 2.1–6.2 s
2 R OT nodular heterotopia (occipital horn). Abnormal overlying cortex over the whole posterior quadrant with increase of thickness R post. T spikes 30 R post. T sharp rhythmic activity
Electrical event
1 22.3 s
3 Bil. nodular heterotopia (trigone). L anterior T resection R T spikes
L T spikes
41
40
R centroT 10Hz activity
Clinical event
1 65 s
4 Bil. nodular heterotopia (trigones, temporal and occipital horns). Thick overlying occipital cortex R T spikes
L T spikes
23
8
L Trhythmic sharp waves
Electrical events
3 2.5 s; 1.6–3.5 s
5 R ant. F focal cortical dysplasia R F spike and waves 31 R F rhythmic spike and waves
Clinical events
15 21.3 s; 7.2– 49.7 s
6 L centro P focal cortical dysplasia L centro P spikes 42 Rhythmic L centro P sharp fast activity
Clinical events
1 9.2 s
7 Band heterotopia predominating over parietal, occipital and temporal areas, R 4L L TO polyspikes
R TO polyspikes
100
74
R TPO discharges propagating to L homologous regions
Electrical events during sleep
11 6.2; 3.8–13 s
8 Band heterotopia predominating over the frontal and parietal regions, bilaterally Bil. TPO rhythmic slow waves 15 Rhythmic bil. occipital and R TPO discharges
Electrical events during sleep
2 12 s; 10.5–13.5
Mean ± SD 38 ± 27.5 7.2 ± 8.6 17.7 ± 20.5 s

R = right; L = left; Bil = bilateral; T = temporal; F = frontal; C = central; P = parietal; O = occipital.